One-liner#

Adult/geriatric approach to a swollen single joint: rapidly separate septic arthritis and fracture/hemarthrosis from gout/pseudogout and non-emergent causes, then choose the right aspiration/imaging pathway.

Quick nav#

Red flags / send to ED#

  • Suspected septic arthritis: fever/systemic illness plus hot swollen joint with severe pain on any motion
  • Suspected fracture/dislocation: major trauma, deformity, inability to bear weight/use the limb
  • Suspected hemarthrosis with significant swelling/pain (especially on anticoagulation) or neurovascular compromise
  • Rapidly worsening pain/swelling with pain out of proportion (rare: compartment syndrome/necrotizing infection)
  • Immunosuppression/IVDU with acute hot swollen joint (lower threshold for ED/urgent aspiration)

Key history#

  • Time course: sudden (hours) vs over days; first episode vs recurrent
  • Trauma: fall/twist; anticoagulation/bleeding disorders (hemarthrosis)
  • Systemic symptoms: fever, chills, malaise
  • Prior gout/pseudogout; recent dehydration/diuretics; recent illness or surgery
  • Risk for infection: prosthetic joint, immunosuppression, diabetes, skin breaks/cellulitis near joint, IVDU
  • Recent GI/GU infection (reactive arthritis), recent tick exposure (regional)
  • Location: knee/ankle/first MTP/wrist are common for crystal disease

Focused exam#

  • Vitals; fever; overall toxicity
  • Inspect: erythema, warmth, swelling distribution; wounds or nearby cellulitis
  • Palpate effusion; compare sides; assess bony tenderness (fracture)
  • ROM: pain with passive ROM suggests intra-articular process (infection/crystal)
  • Stability testing (if trauma) as tolerated
  • Neurovascular exam distal to joint

Differential (quick pattern recognition)#

Common/likely (outpatient)#

DiagnosisKeywords patients useKey cluesFocused exam cluesInitial next step
Gout flare“Sudden,” “throbbing,” “even a sheet hurts”Rapid onset; prior episodes; diuretics/dehydrationHot swollen joint; extreme tendernessTreat flare if infection unlikely; aspirate if uncertain
Pseudogout (CPPD)“Sudden swollen knee/wrist”Older; knee/wrist common; may follow illnessEffusion; warmthTreat flare; aspirate if uncertain
Traumatic effusion/sprain“Twisted it,” swelling after injuryClear trauma; swelling may be delayedEffusion; ligament tendernessX-ray if indicated; brace/rehab
OA flare with effusion“Ache,” “stiff,” worse after activityKnown OA; less dramatic onsetMild–moderate effusion; ROM limited by painConservative care; consider aspiration if tense/unclear
Bursitis (prepatellar/olecranon)“Soft lump,” “swollen over kneecap/elbow”Localized swelling over bursaFluctuant superficial swelling; joint ROM relatively preservedCompression/avoid pressure; aspirate selectively
Hemarthrosis (anticoagulation)“Blew up/swollen fast”On anticoagulant; minor traumaLarge effusion; bruisingUrgent evaluation; consider aspiration/imaging

Can’t-miss / urgent#

DiagnosisKeywords patients useKey cluesFocused exam cluesInitial next step
Septic arthritis“Hot,” “feels sick,” “can’t move it”Fever/risk factors; rapidly progressiveSevere pain with passive ROM; effusionED now; aspiration/cultures + antibiotics per protocol
Fracture/dislocation“Can’t bear weight,” deformityTrauma; focal bony painBony tenderness/deformityED/urgent imaging + immobilize
Necrotizing infection/compartment syndrome (rare)“Pain out of proportion”Rapid progression; systemic signsTense swelling; severe painED now

Workup#

  • Aspirate when:
    • The diagnosis is unclear and infection is in the differential, or
    • The presentation is atypical for known gout/CPPD, or
    • There is a large tense effusion limiting ROM.
  • Typical synovial fluid studies (verify local protocol): cell count/diff, crystals, Gram stain/culture (and consider glucose/protein if used locally).
  • Do not rely on serum uric acid during an acute flare to rule in/out gout.
  • X-ray for trauma, deformity, focal bony tenderness, suspected OA, or to evaluate chondrocalcinosis (CPPD).
  • Labs (when indicated): CBC + ESR/CRP for suspected infection/inflammatory disease; blood cultures if septic arthritis suspected.

Synovial fluid interpretation (quick reference)#

FindingNormalNon-inflammatory (OA)Inflammatory (gout/RA)Septic
WBC count<200/μL200–2,000/μL2,000–50,000/μL>50,000/μL (often >100k)
PMN %<25%<25%>50%>75%
AppearanceClearClear/yellowCloudy/yellowPurulent
CrystalsNoneNoneUrate (gout) or CPPDUsually none

Key points:

  • WBC >50,000 with >75% PMNs: treat as septic until proven otherwise
  • Crystal-positive does NOT rule out infection (can coexist)
  • Gram stain is only ~50% sensitive for septic arthritis—do not rely on negative Gram stain to exclude infection

Initial management#

  • If septic arthritis is possible: prioritize urgent aspiration/ED pathway and antibiotics per local protocol (do not delay).
  • If crystal flare is likely and infection is unlikely: treat with anti-inflammatory therapy based on comorbidities—see medication tables below.
  • Protect the joint: relative rest, ice, compression, elevation; consider bracing if traumatic instability suspected.

Anti-inflammatory options for crystal arthritis flares#

DrugDoseContraindicationsMonitoringCostNotes
Naproxen500 mg BID × 5–7 daysCKD (eGFR <30), GI bleed/ulcer, HF, anticoagulationCr if CKD risk$First-line if no NSAID contraindications
Indomethacin50 mg TID × 2 days, then 25 mg TID × 3 daysSame as naproxen; higher GI/CNS side effectsSame as naproxen$Potent but more side effects; avoid in elderly
Colchicine1.2 mg, then 0.6 mg 1 hour later (day 1); then 0.6 mg daily–BIDCKD (reduce dose if eGFR <30); hepatic impairment; CYP3A4 inhibitorsGI symptoms (diarrhea); Cr$$Most effective within 24 hours of flare onset; low-dose regimen preferred
Prednisone30–40 mg daily × 5 days (no taper needed)Uncontrolled DM, active infectionBlood glucose in diabetics$Use when NSAIDs/colchicine contraindicated; safe in CKD
Intra-articular triamcinolone10–40 mg depending on joint sizeActive infection; overlying cellulitisBlood glucose in diabetics$Excellent for monoarticular flare; avoids systemic side effects

Choosing therapy:

  • No contraindications: NSAID (naproxen preferred) or colchicine
  • CKD/HF/GI risk: Prednisone or intra-articular steroid
  • Diabetes: NSAID or colchicine preferred (steroids spike glucose)
  • Monoarticular with secure diagnosis: Intra-articular steroid is highly effective

When NOT to treat empirically:

  • Fever or systemic illness (aspirate first to rule out infection)
  • First presentation without prior gout diagnosis (consider aspiration)
  • Prosthetic joint (always aspirate; do not inject without orthopedic guidance)

Management by diagnosis#

Gout flare (likely; infection unlikely)#

  • Education: gout can mimic infection; new patterns or systemic symptoms should prompt aspiration/urgent evaluation.
  • Treatment: anti-inflammatory flare therapy per patient risk profile (NSAID/colchicine/steroid options; verify local protocol); avoid starting urate-lowering changes mid-flare unless already on therapy.
  • Follow-up: 48–72 hours if not improving; earlier if fever, spreading redness, or worsening pain.

Pseudogout (CPPD)#

  • Education: common in older adults; often knee or wrist; can recur.
  • Treatment: anti-inflammatory therapy; consider aspiration/injection pathway when diagnosis is secure and severe (verify local protocol).
  • Follow-up: 3–7 days if not improving.

Traumatic effusion/sprain#

  • Education: swelling can lag injury; focus on stability and function.
  • Treatment: ice/compression/elevation, brace as indicated, early ROM/rehab once fracture ruled out; image based on location-specific rules.
  • Follow-up: 1–2 weeks to reassess stability and ROM.

Suspected septic arthritis#

  • Education: joint infection is time-sensitive and can permanently damage the joint.
  • Treatment: ED now for aspiration/cultures and antibiotics per protocol; do not treat as gout without ruling out infection when uncertain.
  • Follow-up: ED pathway.

Follow-up#

  • If diagnosis is uncertain or symptoms are severe: reassess in 24–72 hours (or sooner) and ensure aspiration/imaging path is clear.
  • For typical non-emergent flares (OA/bursitis/known crystal): reassess in 1–2 weeks if not improving.
  • Escalate urgently for fever, rapidly increasing redness/swelling, inability to move the joint, or new neurovascular symptoms.

Patient instructions#

  • Rest the joint for the next 1–2 days, but keep gentle range of motion as tolerated.
  • Use ice 10–15 minutes at a time and consider compression/elevation for swelling.
  • Use over-the-counter pain options if safe for you.
  • Seek urgent care now for fever, rapidly spreading redness, severe pain with any joint movement, or inability to bear weight/use the limb.

Smartphrase snippets#

Gout flare, treating empirically: Acute monoarticular swelling of [joint] consistent with gout flare. No fever, no systemic symptoms, prior gout history. Infection unlikely. Treating with [NSAID/colchicine/prednisone]. Return precautions for fever, spreading redness, or worsening pain reviewed. Follow-up in 48–72 hours if not improving.

Aspiration performed: [Joint] aspirated for diagnostic evaluation of acute monoarticular swelling. [X mL] of [cloudy/clear/bloody] fluid obtained. Sent for cell count, crystals, Gram stain, and culture. [If injected: Injected with triamcinolone X mg after aspiration.] Pending results; patient instructed to return immediately for fever or worsening symptoms.

Referred to ED for septic arthritis concern: Acute hot swollen [joint] with [fever/systemic symptoms/severe pain with passive ROM]. Septic arthritis cannot be excluded. Referred to ED for urgent aspiration, cultures, and IV antibiotics. Patient instructed to go directly to ED.

Complaint pages#

  • Polyarthralgia — multiple joint involvement and inflammatory arthritis screening
  • Ankle Pain — ankle-specific evaluation including gout
  • Knee Pain — knee-specific evaluation and effusion management

Problem pages#

  • Gout — comprehensive gout management including flare treatment and urate-lowering therapy
  • Osteoarthritis — OA management when effusion is secondary to degenerative disease